For some reason the world health communities are turning a blind eye to the hazards of 5G. The global censorship regarding the impact of 5G on our body is outrages. Aren’t we all made out of flesh and blood?
During the recent years countless doctors fought alone to stop 5G as it is not just another communication technology. They failed.
5G will be used in hospitals while it is not healthy in itself. EMF is simply not healthy never the less 5G.
This multi billions industry put technology first and health -last.
Will evil win? The health forecasts for new oncologists patients were already published in countries where 5G is installed. They never link it to 5G. It is an invisible enemy of the human race. Why do we accept it?
Exposed: “I can’t breath” : EMF .. increased the rate of deoxygenation – experiments New York
Non-Thermal Radio Frequency and Static Magnetic Fields Increase Rate of Hemoglobin Deoxygenation in a Cell-Free Preparation
David Muehsam, 1 , 3 , * Parviz Lalezari, 2 Rukmani Lekhraj, 1 Provvidenza Abruzzo, 5 Alessandra Bolotta, 5 Marina Marini, 5 Ferdinando Bersani, 6 , 7 Giorgio Aicardi, 3 , 7 Arthur Pilla, 4 , 8 and Diana Casper 1
The growing body of clinical and experimental data regarding electromagnetic field (EMF) bioeffects and their therapeutic applications has contributed to a better understanding of the underlying mechanisms of action. This study reports that two EMF modalities currently in clinical use, a pulse-modulated radiofrequency (PRF) signal, and a static magnetic field (SMF), applied independently, increased the rate of deoxygenation of human hemoglobin (Hb) in a cell-free assay. Deoxygenation of Hb was initiated using the reducing agent dithiothreitol (DTT) in an assay that allowed the time for deoxygenation to be controlled (from several min to several hours) by adjusting the relative concentrations of DTT and Hb. The time course of Hb deoxygenation was observed using visible light spectroscopy. Exposure for 10–30 min to either PRF or SMF increased the rate of deoxygenation occurring several min to several hours after the end of EMF exposure. The sensitivity and biochemical simplicity of the assay developed here suggest a new research tool that may help to further the understanding of basic biophysical EMF transduction mechanisms. If the results of this study were to be shown to occur at the cellular and tissue level, EMF-enhanced oxygen availability would be one of the mechanisms by which clinically relevant EMF-mediated enhancement of growth and repair processes could occur.